Early Childhood, Neural Imprinting, and Non-Hypnotic Regression
- Iliyana Petrova
- 4 days ago
- 4 min read
What Denmark’s Shift Away from “Cry It Out” Teaches Us About Healing Later in Life
Early childhood is more than a developmental phase—it is a neurobiological foundation for emotional regulation, stress response, attachment formation, and later cognitive and social outcomes. Modern neuroscience increasingly confirms what attachment theory and developmental psychology have long asserted: the infant brain is exquisitely sensitive to relational experiences.
This understanding has now reached national policy level in Denmark.
Denmark’s Landmark Decision: Science Over Tradition
Denmark is officially moving away from the “cry it out” approach after a nationwide study revealed it was still being taught across municipalities and despite substantial professional concern. In response, over 700 psychologists signed a unified statement calling for immediate discontinuation, emphasizing that elevated stress hormones and disrupted caregiver-infant attunement can risk long-term neural and emotional outcomes.
While debates persist about the specific outcomes of “cry it out” sleep training in infancy (some longitudinal studies find no direct adverse effects on attachment measured at 18 months), these findings do not fully address the neuroendocrine mechanisms underlying stress responses and long-term regulation strategies in early life.
The Biology of Infant Stress and Caregiver Response
Infant cries are not arbitrary noise—they are biologically meaningful signals. When an infant’s distress is met with caregiver comfort, regulators such as heart rate and breathing are stabilized, and pathways essential for adaptive stress response are reinforced. Sensitive caregiving functions as an external regulator of the infant’s developing autonomic and neuroendocrine systems.
Conversely, cortisol, a core stress hormone produced via the hypothalamic-pituitary-adrenal (HPA) axis, rises significantly in infants experiencing distress, especially when caregiver responses are inconsistent or absent. Research in naturalistic contexts shows caregiving behavior modulates cortisol reactivity, and repeated stressor exposure without soothing can influence the developing brain’s regulation systems.
Attachment and Neurodevelopment: What the Evidence Shows
Infant attachment security is shaped by caregiver responsiveness, which affects stress hormone patterns and later emotional functioning. For example:
Secure attachment patterns correlate with lower cortisol elevations during stress, while insecure patterns align with heightened physiological stress responses.
Sensitive caregiving supports early neurodevelopment and emotional regulation, programming circuits in regions such as the amygdala, prefrontal cortex, and hippocampus—areas central to emotion and stress processing.
These connections between early experience and neurodevelopment illustrate why responsive caregiving matters for long-term emotional stability—not merely immediate behavior.
The Debate about “Cry It Out”
One longitudinal study of 178 infants reported no adverse developmental or attachment outcomes at 18 months associated with occasional use of “cry it out.” However, researchers and clinicians caution that absence of findings at 18 months does not equate to evidence of no effect on neurobiological systems, especially when key measures like cortisol dynamics and quality of caregiver regulation aren’t directly assessed.
Other work on the neurobiology of parental response shows that infant crying engages multiple interacting systems (hormonal, neural, motivational), underscoring the complexity of interpreting stress and attachment responses in isolation.
The Correlation: Early Emotional Imprinting and Later Patterns
Many adults struggling with:
chronic anxiety
emotional suppression
over-dependency or avoidance in relationships
challenges with trust and regulation
may be operating from implicit nervous system adaptations rooted in early relational patterns—predating conscious memory formation.
These patterns often reflect how the nervous system learned to manage stress, not a flaw in character or intention.
Non-Hypnotic Regression as a Repair Pathway
Non-hypnotic regression processes approach early imprints not by reliving trauma, but by accessing preverbal sensorimotor and affective patterns stored in the nervous system. This can help:
identify early-formed stress and attachment patterns
safely reorganize responses within a relational therapeutic context
establish corrective experiences of safety and containment
Such approaches recognize the plasticity of the nervous system across the lifespan and aim to foster regulation rather than suppression.
Supporting Nervous System Repair Later in Life
Research-aligned practices that support healing and regulatory integration include:
somatic and mindfulness-based regulation practices
attachment-focused therapeutic models
neurodevelopmentally informed interventions
relational experiences that validate emotional expression
These strategies do not erase early experience, but reshape the nervous system’s expectations for safety and co-regulation.
References & Research Sources
Gunnar, M. R., & Donzella, B. (2002)
Social regulation of the cortisol levels in early human development
Psychoneuroendocrinology, 27(1–2), 199–220
https://doi.org/10.1016/S0306-4530(01)00045-2
https://www.sciencedirect.com/science/article/pii/S0306453001000452
Gunnar, M. R., Brodersen, L., Nachmias, M., Buss, K., & Rigatuso, J. (1996)
Stress reactivity and attachment security
Developmental Psychobiology, 29(3), 191–204
https://doi.org/10.1002/(SICI)1098-2302(199604)29:3<191::AID-DEV1>3.0.CO;2-M
Bernard, K., Dozier, M., Bick, J., & Gordon, M. K. (2015)
Intervening to enhance cortisol regulation among children at risk for neglect
Development and Psychopathology, 27(1), 163–174
Tottenham, N. (2012)
Human amygdala development in the absence of species-expected caregiving
Developmental Psychobiology, 54(6), 598–611
Meaney, M. J. (2001)
Maternal care, gene expression, and the transmission of individual differences in stress reactivity
Annual Review of Neuroscience, 24, 1161–1192
Swain, J. E., Lorberbaum, J. P., Kose, S., & Strathearn, L. (2007)
Brain basis of early parent-infant interactions
Journal of Child Psychology and Psychiatry, 48(3–4), 262–287
Middlemiss, W., Granger, D. A., Goldberg, W. A., & Nathans, L. (2012)
Asynchrony of mother-infant hypothalamic-pituitary-adrenal axis activity following extinction of infant crying responses
Early Human Development, 88(4), 227–232
Bilgin, A., & Wolke, D. (2020)
Parental use of “cry it out” in infancy and child attachment and behavioral development
Journal of Child Psychology and Psychiatry, 61(11), 1184–1193
National Scientific Council on the Developing Child (Harvard University)
Excessive stress disrupts the architecture of the developing brain
Tronick, E. Z. (2007)
The Neurobehavioral and Social-Emotional Development of Infants and Children
W.W. Norton & Company
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